Researchers in California just demonstrated that a mere six days of eating all organic is enough to significantly reduce levels of harmful pesticides in your body. In the study, four families (with completely different backgrounds) consumed conventional products for six days and had their urine tested. Then, they ate a 100 percent organic diet for six days and had their urine tested again. The drop in pesticide levels present in their urine between tests was massive.
There has been a recent investigation here on Bellingcat, of a poisoning incident in Bulgaria in April 2015, where an individual named Emilian Gebrev was posioned. An angle of this investigation has been to see if it is at all connected to the attempted murder of Sergei Skripal. One particular aspect of recent publicity has been to examine whether or not a “Novichok” series nerve agent was responsible for the 2015 poisoning incident.
A new peer-reviewed study shows that eating a completely organic diet—even for just one week—can dramatically reduce the presence of pesticide levels in people, a finding that was characterized as “groundbreaking” by critics of an industrial food system that relies heavily on synthetic toxins and chemicals to grow crops and raise livestock. Published in the Environmental Research, the study—titled Organic Diet Intervention Significantly Reduces Urinary Pesticide Levels in U.S.
An Ultra performance liquid chromatography (UPLC) coupled to UV detection method was developed to determine acetamiprid residues in water reservoirs of northern Benin, close to cotton fields. The quantification limit of this method was 0.2 µg L−1 acetamiprid in water, its precision ranged between 8% and 22%, and its trueness between 99% and 117% (for concentrations ranging from 0.2 to 5.0 µg L−1). Acetamiprid residues were determined in water samples collected in four reservoirs from northern Benin during the phytosanitary treatment period of cotton.
Imidacloprid is a widely used insecticide with high runoff potential posing a significant threat to aquatic ecosystems. In order to determine the spatial and temporal concentrations of imidacloprid in Forester Creek, a tributary to the San Diego River, surface water samples were collected from two sites under wet-weather and dry-weather conditions. Imidacloprid was detected with 100% frequency in surface water samples from Forester Creek with a median concentration of 16.9 ng/L (range: 3.8–96.8 ng/L). Over 60% of samples exceeded U.S. EPA's chronic exposure benchmark (10 ng/L).
Yet again, our government scientists—the oft neglected but so important brain trust of our Nation—bring the public some very important new data. Pesticide water monitoring experts at the U.S. Geological Survey (USGS) paired up with scientists from the University of Iowa in a federally-funded collaboration to track neonicotinoid pesticides or “neonics” in tap water, including the potential to form chlorinated disinfection byproducts (DBPs) from the pesticides and their metabolites that may be more toxic than the original compounds. And, the news isn’t good.
Pesticide use is driving an “alarming” decline in the world’s insects that could have a “catastrophic” impact on nature’s ecosystems, researchers have warned. More than 40 per cent of insect species are at risk of extinction with decades, with climate change and pollution also to blame, according to a global scientific review. Their numbers are plummeting so precipitously that almost all insects could vanish within a century, the study found.
Imidacloprid - a type of neonicotinoid - changes the way that worker bees interact with the colony’s larvae: they become less social, stop nursing larvae, experience altered social and spatial dynamics within nests, and cease hive insulation construction. A research team led by James Crall of Harvard University investigated the effects of imidacloprid using a robotic platform for continuous, multicolony monitoring of uniquely identified workers. Their research showed that the behaviours induced by imidacloprid lead to colony collapse.
We previously determined a metabolite of acetamiprid, N-desmethyl-acetamiprid in the urine of a patient, who exhibited some typical symptoms including neurological findings. We sought to investigate the association between urinary N-desmethyl-acetamiprid and the symptoms by a prevalence case-control study. Spot urine samples were collected from 35 symptomatic patients of unknown origin and 50 non-symptomatic volunteers (non-symptomatic group, NSG, 4–87 year-old).